Auflistung nach Autor "Mir, Perihan"

Anzeige der Dokumente 1-12 von 12

A topological refactoring design strategy yields highly stable granulopoietic proteins

Skokowa, Julia; Ritter, Malte; Nasri, Masoud; Xu, Yun; Mir, Perihan; Krahl, Ann-Christin; Rogers, Katherine W.; Maksymenko, Kateryna; Bajoghli, Baubak; Welte, Karl; ElGamacy, Mohammad; Haaf, Jérémy; Aghaallaei, Nargessadat; Müller, Patrick (2022)
CRISPR/Cas9-mediated ELANE knockout enables neutrophilic maturation of primary hematopoietic stem and progenitor cells and induced pluripotent stem cells o severe congenital neutropenia patients

Nasri, Masoud; Ritter, Malte; Mir, Perihan; Dannenmann, Benjamin; Aghaallaei, Nargessadat; Amend, Diana; Xu, Yun; Bernhard, Regine; Steiert, Ingeborg; Hähnel, Karin; Berger, Jürgen; Koch, Iris; Sailer, Brigitte; Hipp, Katharina; Klimiankou, Maksim; Bajoghli, Baubak; Welte, Karl; Skokowa, Julia (2020)
Design of novel granulopoietic proteins by topological rescaffolding

Hernandez Alvarez, Birte; Skokowa, Julia; Coles, Murray; Mir, Perihan; Nasri, Masoud; Maksymenko, Kateryna; Weidmann-Krebs, Laura; Rogers, Katherine W.; Welte, Karl; Lupas, Andrei N.; ElGamacy, Mohammad; Müller, Patrick (2020)
Fluorescent labeling of CRISPR/Cas9 RNP for gene knockout in HSPCs and iPSCs reveals an essential role for GADD45b in stress response

Nasri, Masoud; Mir, Perihan; Dannenmann, Benjamin; Amend, Diana; Skroblyn, Tessa; Xu, Yun; Schulze-Osthoff, Klaus; Klimiankou, Maksim; Welte, Karl; Skokowa, Julia (2019)
GADD45β plays an essential role in the G-CSF triggered granulocytic differentiation of human hematopoietic stem cells

Mir, Perihan (2022-01-01)

Congenital neutropenia (CN) is a bone marrow failure syndrome caused by inherited gene mutations in e.g., ELANE or HAX1 leading to markedly low neutrophil numbers in peripheral blood. The mechanism of the “maturation arrest” ...
GADD45β plays an essential role in the G-CSF triggered granulocytic differentiation of human hematopoietic stem cells

Mir, Perihan (2020)
GM-CSF treatment is not effective in congenital neutropenia patients due to its inability to activate NAMPT signaling

Samareh, Bardia; Morishima, Tatsuya; Mir, Perihan; Kanz, Lothar; Skokowa, Julia; Welte, Karl (2017)
Human iPSC-based model of severe congenital neutropenia reveals elevated UPR and DNA damage in CD34(+) cells preceding leukemic transformation

Dannenmann, Benjamin; Zahabi, Azadeh; Mir, Perihan; Oswald, Benedikt; Bernhard, Regine; Klimiankou, Maksim; Morishima, Tatsuya; Schulze-Osthoff, Klaus; Kanz, Lothar; Welte, Karl; Skokowa, Julia (2019)
iPSC modeling of stage-specific leukemogenesis reveals BAALC as a key oncogene in severe congenital neutropenia

Dannenmann, Benjamin; Klimiankou, Maksim; Oswald, Benedikt; Solovyeva, Anna; Mardan, Jehan; Nasri, Masoud; Ritter, Malte; Zahabi, Azadeh; Arreba-Tutusaus, Patricia; Mir, Perihan; Stein, Frederic; Morishima, Tatsuya; Welte, Karl; Skokowa, Julia (2021)
Molecular and functional interactions between AKT and SOX2 in breast carcinoma

Wang, Hui; Mir, Perihan; Merz, Britta; Kanz, Lothar; Schulze-Osthoff, Klaus; Lengerke, Claudia; Rothfuss, Oliver (2015)
NAMPT/SIRT2-mediated inhibition of the p53-p21 signaling pathway is indispensable for maintenance and hematopoietic differentiation of human iPS cells

Xu, Yun; Nasri, Masoud; Dannenmann, Benjamin; Mir, Perihan; Zahabi, Azadeh; Welte, Karl; Morishima, Tatsuya; Skokowa, Julia (2021)
New insights into the pathomechanism of cyclic neutropenia

Mir, Perihan; Klimiankou, Maksim; Skokowa, Julia; Welte, Karl; Findik, Betül; Hähnel, Karin (2020)

Anzeige der Dokumente 1-12 von 12

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Auflistung nach Autor "Mir, Perihan"

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